De Michele et al.: "Evidence of SARS-CoV-2 spike protein on retrieved thrombi from COVID-19 patients"; indicates that SARS-CoV-2 Spike Protein may drive blood clots
"found SARS-CoV-2 Spike protein directly attached to platelets within the blood retrieved clots"
SOURCE:
The key finding is that the SARS-CoV-2 spike protein latches on directly to the platelets in the blood clots taken from patients. This minus the full entire virus. So the spike protein functions on its own in the blood clotting sequelae. One could reasonably speculate that the other vaccine delivery platforms e.g. mRNA lipid-nano-particle, adeno-viral vector etc. does the same in terms of the spike protein (disassociated free spike).
‘The pathophysiology of COVID-19-associated coagulopathy is complex and not fully understood. SARS-CoV-2 spike protein (SP) may activate platelets and interact with fibrin(ogen). We aimed to investigate whether isolated SP can be present in clots retrieved in COVID-19 patients with acute ischemic stroke (by mechanical thrombectomy) and myocardial infarction. In this pilot study, we could detect SP, but not nucleocapsid protein, on platelets of COVID-19 patients’ thrombi. In addition, in all three COVID-19 thrombi analyzed for molecular biology, no SARS-CoV-2 RNA could be detected by real-time polymerase chain reaction. These data could support the hypothesis that free SP, besides the whole virus, may be the trigger of platelet activation and clot formation in COVID-19.’
This statement “we could detect SP, but not nucleocapsid protein, on platelets of COVID-19 patients’ thrombi” is critical. SP (spike protein) alone on platelets of COVID-19 patients’ thrombi suggests this is due to vaccine and not natural infection. If nucleocapsid protein is detected (with SP), then this suggests natural infection (natural immunity). This raises the distinct probability that the spike protein (certainly via the vaccine) is pathological and involved with the blood clotting sequelae.
Dr. Ryan Cole, along with many embalmers, are also observing these horrific "clots" post-mortem. Dr. Ryan Cole explains these structures have a lot of amyloid protein embedded in them, which make them more "rubbery" and fibrous in appearance. The other issue that we need to verify is if spike protein and mRNA can be "shed" onto non-vaccinated people, thereby causing similar issues and pathogenicity. The spike protein load and its deleterious effects seem to be much more pronounced in the vaccinated. We must continue to pray and cry out to God for His mercy for countless numbers of affected people. God bless you, Paul, and thank you for all that you do!!
Makes perfect sense. SP is similar to ACE2 receptor in blood vessels. Autoimmune reaction to continuous MRNA mediated SP production cross reacts with ACE2 and damages blood vessels with resulting thrombi leading to clots.