Patterson et al. showed COVID spike protein (virus infection & thus vaccine) persisting in CD16+ Monocytes in Post-Acute Sequelae of COVID-19 (PASC) up to 15 Months (at least 15 months)
This implies the need to detoxify and dissolve spike protein to prevent it from doing damage especially to the vasculature; post-acute sequelae (PASC) of COVID may affect up to 30-40% of all infected
https://www.frontiersin.org/articles/10.3389/fimmu.2021.746021/full
‘investigated the presence of SARS-CoV-2 S1 (sub-unit) protein in 46 individuals.
analyzed T-cell, B-cell, and monocytic subsets in both severe COVID-19 patients and in patients with post-acute sequelae of COVID-19 (PASC).
The levels of both intermediate (CD14+, CD16+) and non-classical monocyte (CD14Lo, CD16+) were significantly elevated in PASC patients up to 15 months post-acute infection compared to healthy controls (P=0.002 and P=0.01, respectively).
A statistically significant number of non-classical monocytes contained SARS-CoV-2 S1 protein in both severe (P=0.004) and PASC patients (P=0.02) out to 15 months post-infection. Non-classical monocytes were sorted from PASC patients using flow cytometric sorting and the SARS-CoV-2 S1 protein was confirmed by mass spectrometry. Cells from 4 out of 11 severe COVID-19 patients and 1 out of 26 PASC patients contained ddPCR+ peripheral blood mononuclear cells, however, only fragmented SARS-CoV-2 RNA was found in PASC patients. No full length sequences were identified, and no sequences that could account for the observed S1 protein were identified in any patient. That non-classical monocytes may be a source of inflammation in PASC warrants further study.’
What is with all these people talking about “dissolving” proteins?
1. You can denature proteins (which is what stomach acid does).
2. You can then cleave peptide bonds in denatured protein (which is what digestive enzymes produced by the pancreas do in the small intestine to break proteins down into individual amino acids and dipeptides for absorption through the small intestine).
3. Inside individual cells, damaged proteins are broken down into amino acids in lysosomes (the reason it’s done in lysosomes is to protect the rest of the cellular proteins from the harsh processes involved).
4. Inside the body but outside the cells, damaged proteins, for instance low density lipoprotein damaged by oxidation, can be scavenged for disposal. In the case of damaged LDL, foam cells in the blood scavenge them and then extravasate between cells of the arterial intimal lining to the space between that lining and the smooth muscle walls of the artery. And then they die. That is the source of arterial plaque.
5. Or foreign proteins comprising bacteria and viruses can be blasted with chemical agents by the immune system and the bits and pieces be scavenged by immune system cells for
A. Presentation by antigen presenting cells to the B and T cells of the adaptive immune system or
B. disposal by internal cellular processes (see 3 above).
But NONE of those processes involves “dissolving” proteins.
And to the extent that proteins are broken down into individual amino acids, those processes necessarily involve very harsh treatment, which is why the processes are sequestered.
It appears this rectification of the spike must be done early on..perhaps IV Chealtion.