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AwakeNotWoke's avatar

Horseman Prof. Hotez connects the dots. SARS-CoV-2 persists and replicates in atheroschlerotic plaque. There are also immune changes triggered by infection. Risk for CV death increases. Soon Hotez will turn his attention to turbocancers.

"This study showing that severe acute respiratory syndrome coronavirus directly infects coronary artery plaques, producing inflammatory substances, really joins the dots and helps our understanding on why we're seeing so much heart disease in COVID patients," Peter Hotez, MD, professor of molecular virology and microbiology at Baylor College of Medicine in Houston, told Medscape.

Asked whether this direct infection of vascular plaques was unique to SARS-CoV-2 or whether this may also occur with other viruses, both Giannarelli and Hotez said they believe this may be a specific COVID effect.

"I wouldn't say it is likely that other viruses infect coronary arteries in this way, but I suppose it is possible," Giannarelli said.

Hotez pointed out that other viruses can cause inflammation in the heart, such as myocarditis. "But I can't think of another virus that stimulates the sequence of events in coronary artery inflammation like we're seeing here.

COVID Coronary Plaque Infection Confirms CV Risk

https://www.medscape.com/viewarticle/997638?ecd=a2a

Laura R.'s avatar

I did everything on my end to work this out, they refuse to return a call or an email. Avoid them like the plague!

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