'A SARS-like cluster of circulating bat…

[Bill Rice, Jr.]

Before they can make the "vaccine," they have to make the virus (or find it). As I understand things, this is/was the whole reason for "gain of function" research.

Also, the military actually took over these operations as part of their bio-weapons program. Before you can have a bio-weapon, you've got to first have the antidote (the "vaccine") that makes sure this weapon (the new virus/disease) won't wipe out all of your troops or country.

But, here, the "vaccine" was the real weapon. Ironically enough, a vaccine wasn't even needed because the virus, while it may have been quite contagious, wasn't "deadly." It was the response to said virus that killed millions of people. That's what was "deadly."

[Dr. Paul Alexander]

BOOM, on target, bulls eye!

[Ryan]

There was a 2019 flu shot that made people sick. That’s why the flu disappeared for 3 years. And why doctors and nurses didn’t treat common cold an flu with antibiotics which are bacterial infection and people became very sick with pneumonia and ended up in the hospital on a ventilator then Remdesivir and if Remdesivir didn’t kill them morphine killed them and after all that TORTURE AND ATTEMPT OF MURDER ON MY FATHER THE FIRST TIME AND HE WOKE UP AND LIVED ANOTHER WEEK BEFORE HE WAS FOUND BLUE THE SECOND TIME WAS THE DEATH SHOT OF FENTANYL !!! And all of this was invented and patented by Ralph Baric. The first outbreak was in Washington State in a nursing home all patients died nurses and doctors didn’t. What was in common with those patients they all had a FLU SHOT !!!

[R U Kidding me.]

I thought Dr David Martin also said they had vaxxes ready to go in 2016 after this announcement? Hmmm. I could be wrong but I thought he said it in different speeches.

Hillary was supposed to win in 2016. Trump won instead. Did this slow their plans? They regrouped and made a new plan on how to get the most from their scam?

Wasn’t Baric running around in 2018 giving speeches about pandemics and how to make money from one?

Regardless…. The level of planning of this fiasco is deep and evil. Never forget what “science” and governments did to us!!!!

[Tammy Malik]

Why arent any of these labs being closed?? Is there even one bill in Congress or the state houses to stop GOF labs??

[DELMAR L GREENLEAF MD]

Yes.. They’re doing gain of function creation.. They are threatening mankind !!! Just like Fauci did.

[Kathleen Janoski]

That paper was written in 2015.

UNC Chapel Hill & Wuhan created the bioweapon.

[Dr. Paul Alexander]

yes, thats why I keep sharing this paper, this paper to me is the research

[Dr. Paul Alexander]

something flowed from this

[Kathleen Janoski]

That paper is the evidence.

[Carol Padgett]

A firing squad execution would be too good for them. Hang them all for the public and their cohorts to see the results of these evil ones’ deeds of true destruction of the masses of humanity. Their slow kill (and immediate kill ) is still murder isn’t it?!

[BigMamaTEA]

damn them ALL!!! The DESTRUCTION of Scientific-Medicine!! #ScientistsForEvil Will burn in HELL.

[BigMamaTEA]

Dr. Paul....I certainly do not have your base of Study....THAT is EXACTLY that "headline" states.

[Ryan]

RALPH BARIC PUBLISHED THIS PAPER IN 2014 IN NATURE MAGAZINE !!!

HE ALREADY MADE HIS AND ROBERT MALONE MRNA VIRUS/VACCINE/FLU SHOT ATTACH TO HUMANIZED MICE AIRWAYS !!!!

NATURE | NEWS

Engineered bat virus stirs debate over risky research

Lab-made coronavirus related to SARS can infect human cells

Declan Butler

12 November 2015

An experiment that created a hybrid version of a bat coronavirus - one related to the virus that causes SARS (severe acute

respiratory syndrome) - has triggered renewed debate over whether engineering lab variants of viruses with possible pandemic

potential is worth the risks.

In an article published in Nature Medicine 1 on 9 November, scientists investigated a virus called SHC014, which is found in horseshoe

bats in China. The researchers created a chimeric virus, made up of a surface protein of SHC014 and the backbone of a SARS virus

that had been adapted to grow in mice and to mimic human disease. The chimaera infected human airway cells - proving that the

surface protein of SHC014 has the necessary structure to bind to a key receptor on the cells and to infect them. It also caused disease

in mice, but did not kill them.

Although almost all coronaviruses isolated from bats have not been able to bind to the key human receptor, SHC014 is not the first that

can do so. In 2013, researchers reported this ability for the first time in a different coronavirus isolated from the same bat population?,

The findings reinforce suspicions that bat coronaviruses capable of directly infecting humans (rather than first needing to evolve in an

intermediate animal host) may be more common than previously thought, the researchers say.

But other virologists question whether the information gleaned from the experiment justifies the potential risk. Although the extent of

any risk is difficult to assess, Simon Wain-Hobson, a virologist at the Pasteur Institute in Paris, points out that the researchers have

created a novel virus that "grows remarkably well" in human cells. "If the virus escaped, nobody could predict the trajectory," he says.

Creation of a chimaera

The argument is essentially a rerun of the debate over whether to allow lab research that increases the virulence, ease of spread or

host range of dangerous pathogens - what is known as 'gain-of-function' research. In October 2014, the US government imposed a

moratorium on federal funding of such research on the viruses that cause SARS, influenza and MERS (Middle East respiratory

syndrome, a deadly disease caused by a virus that sporadically jumps from camels to people).

The latest study was already under way before the US moratorium began, and the US National Institutes of Health (NIH) allowed it to

proceed while it was under review by the agency, says Ralph Baric, an infectious-disease researcher at the University of North

Carolina at Chapel Hill, a co-author of the study. The NIH eventually concluded that the work was not so risky as to fall under the

moratorium, he says.

But Wain-Hobson disapproves of the study because, he says, it provides little benefit, and reveals little about the risk that the wild

SHC014 virus in bats poses to humans.

Other experiments in the study show that the virus in wild bats would need to evolve to pose any threat to humans - a change that

may never happen, although it cannot be ruled out. Baric and his team reconstructed the wild virus from its genome sequence and

found that it grew poorly in human cell cultures and caused no significant disease in mice.

"The only impact of this work is the creation, in a lab, of a new, non-natural risk," agrees Richard Ebright, a molecular biologist and

biodefence expert at Rutgers University in Piscataway, New Jersey. Both Ebright and Wain-Hobson are long-standing critics of gain-of-

function research.

In their paper, the study authors also concede that funders may think twice about allowing such experiments in the future. "Scientific

review panels may deem similar studies building chimeric viruses based on circulating strains too risky to pursue," they write, adding

that discussion is needed as to "whether these types of chimeric virus studies warrant further investigation versus the inherent risks Useful research

But Baric and others say the research did have benefits. The study findings "move this virus from a candidate emerging pathogen to a

clear and present danger", says Peter Daszak, who co-authored the 2013 paper. Daszak is president of the EcoHealth Alliance, an

international network of scientists, headquartered in New York City, that samples viruses from animals and people in emerging-

diseases hotspots across the globe.

Studies testing hybrid viruses in human cell culture and animal models are limited in what they can say about the threat posed by a wild

virus, Daszak agrees. But he argues that they can help indicate which pathogens should be prioritized for further research attention.

Without the experiments, says Baric, the SHC014 virus would still be seen as not a threat. Previously, scientists had believed, on the

basis of molecular modelling and other studies, that it should not be able to infect human cells. The latest work shows that the virus

has already overcome critical barriers, such as being able to latch onto human receptors and efficiently infect human airway cells, he

says. "I don't think you can ignore that." He plans to do further studies with the virus in non-human primates, which may yield data more

relevant to humans.

Nature | doi:10.1038/nature.2015.18787

References

1. Menachery, V. D. et al. Nature Med. http://dx.doi.org/10.1038/nm.3985 (2015).

2. Ge, X-Y. et al. Nature 503, 535-538 (2013).

[Crixcyon]

100% LIES and always will be forever and ever.