Gill et al.: "Autopsy Histopathologic Cardiac Findings in 2 Adolescents Following Second COVID-19 Vaccine Dose"; Myocarditis in particularly teenage boys has been reported following 2nd Pfizer dose
Gill et al.: "Autopsy Histopathologic Cardiac Findings in 2 Adolescents Following Second COVID-19 Vaccine Dose"; Myocarditis in particularly teenage boys has been reported following 2nd Pfizer dose
The myocardial injury seen in these post COVID vaccine hearts is different from typical myocarditis & has an appearance most closely resembling a catecholamine-mediated stress (toxic) cardiomyopathy.
Please excuse the rather naive question, but why is anyone labeling cardiomyopathy, myocarditis or pericarditis mild? Is it ever truly “mild” in young men or anyone for that matter? What once wasn’t now is and if I’m not mistaken, always leads to a shorter lifespan, a lower quality of life and one where the individual is required to take drugs and have regular cardiology exams for life. Sounds more like a business model than a health benefit to me.
alpha 7 nicotinic acetylcholine receptors - are in the adrenal glands. Chimeric spike S1 blocks nAChRs. That may be leading to increased catecholamine production and various other effects.
Nicotinic Receptor Alpha7 Expression during Mouse Adrenal Gland Development
"The transient expression of α7 during adrenal gland development and its prominent co-expression by a subset of NE chromaffin cells in the adult suggests that the α7 receptor contributes to multiple aspects of adrenal gland development and function that persist into adulthood."
Nicotine lozenges would likely be protective. When faced with a cholinergic paralytic, grab an agonist. Yes, things are bad, can we ever talk about solutions?
Right Cervical Vagotomy Aggravates Viral Myocarditis in Mice Via the Cholinergic Anti-inflammatory Pathway
"Recently, we have also demonstrated that cholinergic stimulation with α7nAChR agonist nicotine had a protective effects in murine viral myocarditis, and selective α7nAchR antagonist methyllycaconitine had a deleterious effects in the same setting (Zheng et al., 2014; Ge et al., 2015, 2016)."
The last sentence of the conclusion of this post reads "The clinical presentation of myocarditis after vaccination was usually mild." My understanding of this observation is that prior to the patients' deaths, there was very little clinical evidence of a life threatening event developing even during the main risk window defined as within the first week following dose #2. Further, the toxic and hyperinflamatory (cytokine storm) evidence are both atypical compared to what is otherwise seen, though still rare, in Myocarditis among teenage males. Is this not smoking gun evidence that pretty much says correlation = causation?
Dr. Alexander, I love everything you post and you often see through a lot of studies. Is there any issues you see with this new study? Because it’s pretty damning for our side.
It is terrible and sad that this continues. These need to be pulled from the market and stopped.
See this Brazilian doc work for a very thorough analysis of the involvement of cathecolamines in post-vaccine myocarditis:
https://pubmed.ncbi.nlm.nih.gov/35971401/
There’s no such thing as “mild” myocarditis per Dr. Peter McCullough.
Please excuse the rather naive question, but why is anyone labeling cardiomyopathy, myocarditis or pericarditis mild? Is it ever truly “mild” in young men or anyone for that matter? What once wasn’t now is and if I’m not mistaken, always leads to a shorter lifespan, a lower quality of life and one where the individual is required to take drugs and have regular cardiology exams for life. Sounds more like a business model than a health benefit to me.
alpha 7 nicotinic acetylcholine receptors - are in the adrenal glands. Chimeric spike S1 blocks nAChRs. That may be leading to increased catecholamine production and various other effects.
Nicotinic Receptor Alpha7 Expression during Mouse Adrenal Gland Development
https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0103861
"The transient expression of α7 during adrenal gland development and its prominent co-expression by a subset of NE chromaffin cells in the adult suggests that the α7 receptor contributes to multiple aspects of adrenal gland development and function that persist into adulthood."
Nicotine lozenges would likely be protective. When faced with a cholinergic paralytic, grab an agonist. Yes, things are bad, can we ever talk about solutions?
Right Cervical Vagotomy Aggravates Viral Myocarditis in Mice Via the Cholinergic Anti-inflammatory Pathway
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5281590/
"Recently, we have also demonstrated that cholinergic stimulation with α7nAChR agonist nicotine had a protective effects in murine viral myocarditis, and selective α7nAchR antagonist methyllycaconitine had a deleterious effects in the same setting (Zheng et al., 2014; Ge et al., 2015, 2016)."
The last sentence of the conclusion of this post reads "The clinical presentation of myocarditis after vaccination was usually mild." My understanding of this observation is that prior to the patients' deaths, there was very little clinical evidence of a life threatening event developing even during the main risk window defined as within the first week following dose #2. Further, the toxic and hyperinflamatory (cytokine storm) evidence are both atypical compared to what is otherwise seen, though still rare, in Myocarditis among teenage males. Is this not smoking gun evidence that pretty much says correlation = causation?
Sharing, thanks
Dr. Alexander, I love everything you post and you often see through a lot of studies. Is there any issues you see with this new study? Because it’s pretty damning for our side.
https://www.news-medical.net/news/20221018/Study-suggests-relationship-between-maternal-COVID-vaccination-SARS-CoV-2-placentitis-and-stillbirth.aspx