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The only way spike proteins persist for years is that they are still being made.

How?

Cellular reverse transcriptase transcribes viral mRNA into a DNA-encoded form and inserts it into the cell’s nuclear DNA. Periodically, just like herpes, something triggers the cell to make mRNA copies of the DNA-encoded viral spike gene.

The result?

The cell detects the presence of viral mRNA and viral protein inside the cells and signals the innate immune system to launch inflammatory attacks to kill those cells.

You cannot “dissolve” a protein any more than you can “dissolve” a virus.

As I said before: What is with all these people talking about “dissolving” proteins?

Here’s what can be done with proteins:

1. You can denature proteins (which is what stomach acid does).

2. You can then cleave peptide bonds in denatured protein (which is what digestive enzymes produced by the pancreas do in the small intestine to break proteins down into individual amino acids and dipeptides for absorption through the small intestine).

3. Inside individual cells, damaged proteins are broken down into amino acids in lysosomes (the reason it’s done in lysosomes is to protect the rest of the cellular proteins from the harsh processes involved).

4. Inside the body but outside the cells, damaged proteins, for instance low density lipoprotein damaged by oxidation, can be scavenged for disposal. In the case of damaged LDL, foam cells in the blood scavenge them and then extravasate between cells of the arterial intimal lining to the space between that lining and the smooth muscle walls of the artery. And then they die. That is the source of arterial plaque.

5. Or foreign proteins comprising bacteria and viruses can be blasted with chemical agents by the immune system and the bits and pieces be scavenged by immune system cells for

A. Presentation by antigen presenting cells to the B and T cells of the adaptive immune system or

B. disposal by internal cellular processes (see 3 above).

But NONE of those processes involves “dissolving” proteins.

And to the extent that proteins are broken down into individual amino acids, those processes necessarily involve very harsh treatment, which is why the processes are sequestered.

Do the effects of the expression of a lone viral gene sound familiar? They should. They sound just like an autoimmune disease.

And the problem with this happening from the viral RNA products?

They are indiscriminate and can do the same cellular reverse transcriptase trick in any cell in any organ system in your body simultaneously.

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Aug 12, 2023·edited Aug 12, 2023

I saw at The Wellness Company site a discussion of mindfulness. Good to see. Taking nattokinase and other supplements should be combined with non-pharmacological interventions, e.g., hacking the vagus nerve. The vagus nerve may be full of viral RNA after SARS-CoV-2 infection. PASC has > 200 symptoms and the current estimate that up to 30% may suffer from PASC could be an underestimate. Symptoms are subjective. There is almost certainly silent or asymptomatic PASC. It may not become symptomatic for years. The vagus nerve is definitely affected. From an article about a recent study:

"The researchers detected SARS-CoV-2 RNA in vagus nerve samples obtained from deceased patients with severe COVID-19 showing direct infection of the nerve was accompanied by inflammatory cell infiltration composed mostly of monocytes—a type of white blood cell that finds and destroys germs and eliminates infected cells. Their analysis revealed a “strong enrichment of genes regulating antiviral responses and interferon signaling,” supporting the idea that vagus nerve inflammation is a common phenomenon with COVID-19.

The researchers also analyzed 23 vagus nerve samples of deceased COVID-19 patients grouped into low, intermediate, and high SARS-CoV-2 RNA viral load to determine if the virus was directly detectable in the vagus nerve and if the viral load correlated with vagus nerve dysfunction. Results showed the virus was present in the vagus nerve and also determined there was a direct correlation between SARS-CoV-2 viral RNA load and dysfunction of the central nervous system.

....

Many long COVID patients are vaccinated, making it difficult to determine whether their prolonged symptoms are due to COVID-19 or vaccine injury. Regardless, both are manifestations of “spike protein-related disease” and share a significant overlap in symptoms, pathogenesis, and treatment, according to the FLCCC.

The FLCCC has developed protocols for people with long COVID and those experiencing post-vaccine injuries, including POTS.

They recommend patients undergo a series of initial tests to determine whether they have long COVID, and a chest scan for those who have respiratory symptoms to differentiate between long COVID and post-vaccine syndrome.

Protocols are geared towards either long COVID (pdf) or post-vaccine injuries (pdf)—including dysautonomia disorders such as POTS—exercised under the care of a practitioner."

COVID-19 Vagus Nerve Inflammation May Lead To Dysautonomia: New Study

https://www.zerohedge.com/political/covid-19-vagus-nerve-inflammation-may-lead-dysautonomia-new-study

Breath of Life: The Respiratory Vagal Stimulation Model of Contemplative Activity

https://www.frontiersin.org/articles/10.3389/fnhum.2018.00397/full

How breathing can help you make better decisions: Two studies on the effects of breathing patterns on heart rate variability and decision-making in business cases

https://www.sciencedirect.com/science/article/abs/pii/S0167876018303258?via%3Dihub

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