SPIKE Protein Detoxifier: COVID gene injection (mRNA) SPIKE Recovery may: help manage amount of spike protein in your body, inhibit spike effects & blood vessel blockage, & support T-cell activity
SPIKE Protein Detoxifier: COVID gene injection (mRNA) SPIKE Recovery may: help manage amount of spike protein in your body, inhibit spike effects & blood vessel blockage, & support T-cell activity
This formulation may potentially help LONG COVID symptoms; The science is maturing & being developed yet people need support now & this formulation may be of benefit; also consider FLCCC's suggestion
Dr Paul, thank you for keeping an open mind. Dr Astrid Stuckelberger was silenced by Dr Ryan Cole at the Swedish Conference during her presentation when she was addressing the graphene oxide and nano-technology which many doctors are finding in the vials. Any remedy that doesn't also address this issue is incomplete, imo.
Hello, this is Rupert Sayre, the associate of The Shadow. I just wanted to write a brief update on the man I'm helping with his Covid mRNA Injection Illness.
At last recount, his elbow was 5% swollen and his feet were becoming increasingly inflamed and swollen.
His elbow healed up completely and his feet continue to become inflamed and swollen but they haven't incapacitated him yet. At the same time, I've observed him holding his head with both hands while sitting hunched over and also holding his head with one hand while lying back. He also lost his balance and fell down again a few days ago.
The mRNA Spike Proteins (I'm assuming) are still circulating in his blood and travelling around causing inflammation. At the same time, he hasn't taken anything to improve his condition.
Now, I have what might be a valuable piece of information. The man had some lab work done around the time when the inflammation and swelling in his hands and feet were ending. We finally got some of the results after delays and while most of his blood chemistry was in the normal range, his Hemoglobin A1C was very high and well outside of the normally accepted range.
Having looked up Hemoglobin A1C, the damage it can cause matched the damage I've seen his blood inflammation create. (1) (2).
I'm also wondering how this might relate to the mRNA Spike Proteins and possibly the infamous Graphene Oxide. Also, if this gives more weight somehow to the theory of Graphene Oxide being negated by Antioxidants.
I also did some microscope research but don't want to also add that here but only a few remarks: I would like to thank "Justice" for informing me about using saline and dyes. I did use saline and the red blood cells maintained their integrity. I could see the individual red blood cells at 1200x magnification. Comparing the man's and my blood, it looked like his had 30% clumping/clotting whereas mine had none. I might have seen a cholesterol plaque in a look at a third sample of the man's blood: A conglomeration of light brown rocks, thin narrow tubes, red blood cell masses -- I'm wondering what this was.
Glycated hemoglobin, also known as HbA1c, glycohemoglobin, hemoglobin A1c, A1C, is a form of hemoglobin (Hb) that is chemically linked to a sugar.
Damage mechanisms
Glycated hemoglobin causes an increase of highly reactive free radicals inside blood cells, altering the properties of their cell membranes. This leads to blood cell aggregation and increased blood viscosity, which results in impaired blood flow.[13]
Another way glycated hemoglobin causes damage is via inflammation, which results in atherosclerotic plaque (atheroma) formation. Free-radical build-up promotes the excitation of Fe2+-hemoglobin through Fe3+-Hb into abnormal ferryl hemoglobin (Fe4+-Hb). Fe4+ is unstable and reacts with specific amino acids in hemoglobin to regain its Fe3+ oxidation state. Hemoglobin molecules clump together via cross-linking reactions, and these hemoglobin clumps (multimers) promote cell damage and the release of Fe4+-hemoglobin into the matrix of innermost layers (subendothelium) of arteries and veins. This results in increased permeability of interior surface (endothelium) of blood vessels and production of pro-inflammatory monocyte adhesion proteins, which promote macrophage accumulation in blood vessel surfaces, ultimately leading to harmful plaques in these vessels.[13]
Highly glycated Hb-AGEs go through vascular smooth muscle layer and inactivate acetylcholine-induced endothelium-dependent relaxation, possibly through binding to nitric oxide (NO), preventing its normal function. NO is a potent vasodilator and also inhibits formation of plaque-promoting LDLs (i.e. “bad cholesterol”) oxidized form.[13]
This overall degradation of blood cells also releases heme from them. Loose heme can cause oxidation of endothelial and LDL proteins, which results in plaques.[13]
Published online 2015 Aug 26. doi: 10.4330/wjc.v7.i8.449
PMCID: PMC4549778
PMID: 26322184
Glycated hemoglobin and its spinoffs: Cardiovascular disease markers or risk factors?
Jumana Saleh
HB GLYCATION AFFECTS BLOOD VISCOSITY AND CONTRIBUTES TO ENDOTHELIAL INFLAMMATION AND VASCULAR DYSFUNCTION
Intracellular glyco-oxidative stress may contribute to vascular endothelial damage through several mechanisms: (1) accumulation of intracellular free radicals alters erythrocyte membrane properties leading to erythrocyte aggregation, increased blood viscosity and impaired blood flow. Shear stress, due to thicker abrasive blood consistency, affecting the vascular endothelium and triggering an inflammatory response that contribute to subsequent atherogenic events[3,4,27,28,30]; (2) buildup of free radicles promotes the oxidation of ferrous Hb (Hb-Fe2+) into ferric Hb (Hb-Fe3+) (methemoglobin), which is further modified, through several oxidation steps, into ferryl hemoglobin (Hb-Fe3+/Fe4+). The ferryl iron (Fe4+) is unstable and regains the Fe3+ state by reacting with specific amino acids in hemoglobin forming covalently cross-linked Hb multimers[31]. The altered Hb structure promotes cellular damage and releases ferryl Hb into the subendothelial matrix. Silva et al[32] demonstrated that ferryl Hb, rather than Hb, or methemoglobin, increased endothelial permeability and production of pro-inflammatory monocyte adhesion proteins that promote macrophage accumulation and a local inflammatory reaction preceding plaque formation; (3) Free Hb penetrates the vascular smooth muscle layer[33] and inactivates endothelium-dependent relaxation induced by acetylcholine[34] possibly through binding to nitric oxide (NO) which is a potent vasodilator which initiates vaso-relaxation in response to stimuli. Nitric oxide also inhibits formation of oxidized LDL[35] which detrimental to endothelial integrity. Inactivation of NO is a major marker of endothelial dysfunction manifested in impaired vasoactive responses[35]. Rodríguez-Mañas et al[36] demonstrated that highly glycosylated Hb inhibited nitric oxide mediated relaxation to a larger extent than low glycated and unglycated Hb. The authors suggested that Hb-AGEs may exacerbate this effect as abundant in vitro and in vivo evidence demonstrates that AGEs inhibit nitric oxide production and function[36]; and (4) Furthermore, accelerated degradation of erythrocytes releases heme which sensitizes endothelial cells to oxidative damage and promotes oxidation of endothelial proteins and low density lipoproteins (LDLs)[31].
Altogether, these adverse modifications trigger a proliferative inflammatory response in the subendothelial space which involves recruitment of a myriad of inflammatory and immune factors including monocytes, platelets, lymphocytes and increased production of various growth factors and cytokines such as IL-1 and TNF-α and adhesion molecules[37]. Oxidized LDL particles are subsequently scavenged by macrophages forming lipid rich foam cells that contribute to the formation of fatty streaks and subsequent build-up of plaque. As atherosclerotic plaque builds up, further insult to the endothelium activates a vicious cycle of inflammatory/oxidation events and further progression of atherosclerosis[38]. The list of endothelial mediators that contribute to this inflammatory/atherogenic process continues to grow. Interleukin-17 (IL-17), produced by T-helper cells, induces chemokines such as IL-6, IFN-γ and TNF-α to recruit monocytes and neutrophils to the site of inflammation. Recent evidence points to additional allergic/hypergic responses, induced by IL-17, which involve cytokines such as IL-8 and eotaxin believed to play a role in atherogenesis. IL-17 induces eotaxin secretion from smooth muscles, macrophages and fat tissue in the atheromatous plaque[39]. The recruitment of eosinophils by eotaxin during the inflammatory process was recently linked to vascular inflammation and cardiovascular disease[40]. Exploring the relation between these inflammatory mediators and oxidative modification of glycated Hb may provide new avenues for understanding the progression of atherogenic events.
THE BIOMEDICAL STATE IN CANADA; JAIL TIME LOSS OF NETWORTH for 'Misinformation'; EXTRAJURIDICAL HEARINGS WITHOUT PROTECTIONS. 'FACT' deemed what they write on a certificate.
Nice looking product. Thanks for sharing. I added your post and the product info to my new webpage Prenatal/Child on https://jenniferdepew.com/prenatal%2Fchild, It is in the third section about Zinc Ionophores - *black seed oil and EGCG are zinc ionophores and dandelion would help with excess heavy metals.
I wish I connected this information with my husband’s symptoms in the hospital. I only was connecting them after the follow up from the clot they found in his cartoroid artery last February. And then when he a bunch of them in last August and September and then passed. Idk if this item would have helped dissolve the clots and stopped his lung from collapsing. I told the drs the clots were from the vaccine and maybe one kind of listened. They didn’t know how to stop the clotting and bleeding! I didn’t understand why they didn’t know how. He couldn’t breathe bc of the clotting in his lungs. He was on a vent and then a Trach. I read something from a dr in a Twitter chat that he knew and was helping stop these clots. If I only knew then and if only Stacy was his patient. :(
Glad I could offer some info. Interesting about your friend and how the HIV meds may have provided protection from the shot. Hmmm.. I have meter had the bioweapon shot or even been stabbed. I too, just want to check my levels of heavy metals from decades of environmental exposure and detox if necessary. It may become a necessary thing for humanity and I want to be on top of it and share info with others. If you haven't, check out Clifford Carnicom's work. He has been studying the effects of chemtrails for decades. I will post a link with he & Dr. Ana Mihalcea discussing w/ slides.
Does anyone know if Gardasil {HPV vaccine) is safe for young boys? Someone just phoned to ask and I have no idea, although I was speaking with someone recently who knows a kid who had seizures after Gardasil. She was very wary of Gardasil despite having had 4 Pfizer covid jabs.
Is it the Spike Protein that causes the premature aging from catching COVID, or some other part of the virus that causes it. The reports vary widely. Some report that each bout of Covid is equivalent to 3-4 years of aging, others report that severe covid causes 20 years of aging of the mmune system and others that covid makes one look visibly older. If the virus causes such dramatic aging, which supposedly is worsened with each reinfection, what do jabs and repeat jabs do?
I purchased 2 bottles 3 months ago. A bit spendy but it's an all in one for ease of adminstration. I have also followed flccc.net protocol and also Dr Zelenko, rip, for almost 2 years. Made little labeled pill pouches with everything in them for a my family members. Also spendy but they all had it on hand when they got covid. So for no one was hospitalized.
Thank you for all the good articles.
Every time I see something on this topic, I’ve been taking note of the links, so I’ll add this one to my list so far:
https://zstacklife.com/products/z-shield
https://coach.mygotodoc.com/phoenix-forty/
https://worldcouncilforhealth.org/resources/spike-protein-detox-guide/
https://open.substack.com/pub/worldcouncilforhealth/p/spike-protein-detox-guide
https://mobile.twitter.com/georgepup949/status/1616859909332828160
https://open.substack.com/pub/palexander/p/spike-protein-detoxifier-covid-gene
https://open.substack.com/pub/palexander/p/spike-protein-detoxifier-covid-gene-e13
Dr Paul, thank you for keeping an open mind. Dr Astrid Stuckelberger was silenced by Dr Ryan Cole at the Swedish Conference during her presentation when she was addressing the graphene oxide and nano-technology which many doctors are finding in the vials. Any remedy that doesn't also address this issue is incomplete, imo.
I also read somewhere about Chelation Therapy using EDTA; does anybody here reading this know more?
Thank you Dr Alexander for yet another excellent article.
Yikes- too pricey for many of us.
Dr Alexander ..... do you earn money from any of your subscribers that buy the spike recovery product?
Hello, this is Rupert Sayre, the associate of The Shadow. I just wanted to write a brief update on the man I'm helping with his Covid mRNA Injection Illness.
At last recount, his elbow was 5% swollen and his feet were becoming increasingly inflamed and swollen.
His elbow healed up completely and his feet continue to become inflamed and swollen but they haven't incapacitated him yet. At the same time, I've observed him holding his head with both hands while sitting hunched over and also holding his head with one hand while lying back. He also lost his balance and fell down again a few days ago.
The mRNA Spike Proteins (I'm assuming) are still circulating in his blood and travelling around causing inflammation. At the same time, he hasn't taken anything to improve his condition.
Now, I have what might be a valuable piece of information. The man had some lab work done around the time when the inflammation and swelling in his hands and feet were ending. We finally got some of the results after delays and while most of his blood chemistry was in the normal range, his Hemoglobin A1C was very high and well outside of the normally accepted range.
Having looked up Hemoglobin A1C, the damage it can cause matched the damage I've seen his blood inflammation create. (1) (2).
I'm also wondering how this might relate to the mRNA Spike Proteins and possibly the infamous Graphene Oxide. Also, if this gives more weight somehow to the theory of Graphene Oxide being negated by Antioxidants.
I also did some microscope research but don't want to also add that here but only a few remarks: I would like to thank "Justice" for informing me about using saline and dyes. I did use saline and the red blood cells maintained their integrity. I could see the individual red blood cells at 1200x magnification. Comparing the man's and my blood, it looked like his had 30% clumping/clotting whereas mine had none. I might have seen a cholesterol plaque in a look at a third sample of the man's blood: A conglomeration of light brown rocks, thin narrow tubes, red blood cell masses -- I'm wondering what this was.
=========
(1)
https://en.m.wikipedia.org/wiki/Glycated_hemoglobin
Glycated hemoglobin, also known as HbA1c, glycohemoglobin, hemoglobin A1c, A1C, is a form of hemoglobin (Hb) that is chemically linked to a sugar.
Damage mechanisms
Glycated hemoglobin causes an increase of highly reactive free radicals inside blood cells, altering the properties of their cell membranes. This leads to blood cell aggregation and increased blood viscosity, which results in impaired blood flow.[13]
Another way glycated hemoglobin causes damage is via inflammation, which results in atherosclerotic plaque (atheroma) formation. Free-radical build-up promotes the excitation of Fe2+-hemoglobin through Fe3+-Hb into abnormal ferryl hemoglobin (Fe4+-Hb). Fe4+ is unstable and reacts with specific amino acids in hemoglobin to regain its Fe3+ oxidation state. Hemoglobin molecules clump together via cross-linking reactions, and these hemoglobin clumps (multimers) promote cell damage and the release of Fe4+-hemoglobin into the matrix of innermost layers (subendothelium) of arteries and veins. This results in increased permeability of interior surface (endothelium) of blood vessels and production of pro-inflammatory monocyte adhesion proteins, which promote macrophage accumulation in blood vessel surfaces, ultimately leading to harmful plaques in these vessels.[13]
Highly glycated Hb-AGEs go through vascular smooth muscle layer and inactivate acetylcholine-induced endothelium-dependent relaxation, possibly through binding to nitric oxide (NO), preventing its normal function. NO is a potent vasodilator and also inhibits formation of plaque-promoting LDLs (i.e. “bad cholesterol”) oxidized form.[13]
This overall degradation of blood cells also releases heme from them. Loose heme can cause oxidation of endothelial and LDL proteins, which results in plaques.[13]
(2)
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4549778/
World J Cardiol. 2015 Aug 26; 7(8): 449–453.
Published online 2015 Aug 26. doi: 10.4330/wjc.v7.i8.449
PMCID: PMC4549778
PMID: 26322184
Glycated hemoglobin and its spinoffs: Cardiovascular disease markers or risk factors?
Jumana Saleh
HB GLYCATION AFFECTS BLOOD VISCOSITY AND CONTRIBUTES TO ENDOTHELIAL INFLAMMATION AND VASCULAR DYSFUNCTION
Intracellular glyco-oxidative stress may contribute to vascular endothelial damage through several mechanisms: (1) accumulation of intracellular free radicals alters erythrocyte membrane properties leading to erythrocyte aggregation, increased blood viscosity and impaired blood flow. Shear stress, due to thicker abrasive blood consistency, affecting the vascular endothelium and triggering an inflammatory response that contribute to subsequent atherogenic events[3,4,27,28,30]; (2) buildup of free radicles promotes the oxidation of ferrous Hb (Hb-Fe2+) into ferric Hb (Hb-Fe3+) (methemoglobin), which is further modified, through several oxidation steps, into ferryl hemoglobin (Hb-Fe3+/Fe4+). The ferryl iron (Fe4+) is unstable and regains the Fe3+ state by reacting with specific amino acids in hemoglobin forming covalently cross-linked Hb multimers[31]. The altered Hb structure promotes cellular damage and releases ferryl Hb into the subendothelial matrix. Silva et al[32] demonstrated that ferryl Hb, rather than Hb, or methemoglobin, increased endothelial permeability and production of pro-inflammatory monocyte adhesion proteins that promote macrophage accumulation and a local inflammatory reaction preceding plaque formation; (3) Free Hb penetrates the vascular smooth muscle layer[33] and inactivates endothelium-dependent relaxation induced by acetylcholine[34] possibly through binding to nitric oxide (NO) which is a potent vasodilator which initiates vaso-relaxation in response to stimuli. Nitric oxide also inhibits formation of oxidized LDL[35] which detrimental to endothelial integrity. Inactivation of NO is a major marker of endothelial dysfunction manifested in impaired vasoactive responses[35]. Rodríguez-Mañas et al[36] demonstrated that highly glycosylated Hb inhibited nitric oxide mediated relaxation to a larger extent than low glycated and unglycated Hb. The authors suggested that Hb-AGEs may exacerbate this effect as abundant in vitro and in vivo evidence demonstrates that AGEs inhibit nitric oxide production and function[36]; and (4) Furthermore, accelerated degradation of erythrocytes releases heme which sensitizes endothelial cells to oxidative damage and promotes oxidation of endothelial proteins and low density lipoproteins (LDLs)[31].
Altogether, these adverse modifications trigger a proliferative inflammatory response in the subendothelial space which involves recruitment of a myriad of inflammatory and immune factors including monocytes, platelets, lymphocytes and increased production of various growth factors and cytokines such as IL-1 and TNF-α and adhesion molecules[37]. Oxidized LDL particles are subsequently scavenged by macrophages forming lipid rich foam cells that contribute to the formation of fatty streaks and subsequent build-up of plaque. As atherosclerotic plaque builds up, further insult to the endothelium activates a vicious cycle of inflammatory/oxidation events and further progression of atherosclerosis[38]. The list of endothelial mediators that contribute to this inflammatory/atherogenic process continues to grow. Interleukin-17 (IL-17), produced by T-helper cells, induces chemokines such as IL-6, IFN-γ and TNF-α to recruit monocytes and neutrophils to the site of inflammation. Recent evidence points to additional allergic/hypergic responses, induced by IL-17, which involve cytokines such as IL-8 and eotaxin believed to play a role in atherogenesis. IL-17 induces eotaxin secretion from smooth muscles, macrophages and fat tissue in the atheromatous plaque[39]. The recruitment of eosinophils by eotaxin during the inflammatory process was recently linked to vascular inflammation and cardiovascular disease[40]. Exploring the relation between these inflammatory mediators and oxidative modification of glycated Hb may provide new avenues for understanding the progression of atherogenic events.
https://lawyerlisa.substack.com/p/misinformation-leading-to-jail-in?utm_source=substack&utm_medium=email
THE BIOMEDICAL STATE IN CANADA; JAIL TIME LOSS OF NETWORTH for 'Misinformation'; EXTRAJURIDICAL HEARINGS WITHOUT PROTECTIONS. 'FACT' deemed what they write on a certificate.
Nice looking product. Thanks for sharing. I added your post and the product info to my new webpage Prenatal/Child on https://jenniferdepew.com/prenatal%2Fchild, It is in the third section about Zinc Ionophores - *black seed oil and EGCG are zinc ionophores and dandelion would help with excess heavy metals.
I think it was Dr. Tyson.
I wish I connected this information with my husband’s symptoms in the hospital. I only was connecting them after the follow up from the clot they found in his cartoroid artery last February. And then when he a bunch of them in last August and September and then passed. Idk if this item would have helped dissolve the clots and stopped his lung from collapsing. I told the drs the clots were from the vaccine and maybe one kind of listened. They didn’t know how to stop the clotting and bleeding! I didn’t understand why they didn’t know how. He couldn’t breathe bc of the clotting in his lungs. He was on a vent and then a Trach. I read something from a dr in a Twitter chat that he knew and was helping stop these clots. If I only knew then and if only Stacy was his patient. :(
RG
Glad I could offer some info. Interesting about your friend and how the HIV meds may have provided protection from the shot. Hmmm.. I have meter had the bioweapon shot or even been stabbed. I too, just want to check my levels of heavy metals from decades of environmental exposure and detox if necessary. It may become a necessary thing for humanity and I want to be on top of it and share info with others. If you haven't, check out Clifford Carnicom's work. He has been studying the effects of chemtrails for decades. I will post a link with he & Dr. Ana Mihalcea discussing w/ slides.
https://open.substack.com/pub/anamihalceamdphd/p/synthetic-biological-life-forms-cross?utm_source=direct&r=e9snc&utm_campaign=post&utm_medium=web
ALSO:
https://open.substack.com/pub/anamihalceamdphd/p/geoengineered-transhumanism-weaponized?utm_source=direct&r=e9snc&utm_campaign=post&utm_medium=web
Does anyone know if Gardasil {HPV vaccine) is safe for young boys? Someone just phoned to ask and I have no idea, although I was speaking with someone recently who knows a kid who had seizures after Gardasil. She was very wary of Gardasil despite having had 4 Pfizer covid jabs.
Is it the Spike Protein that causes the premature aging from catching COVID, or some other part of the virus that causes it. The reports vary widely. Some report that each bout of Covid is equivalent to 3-4 years of aging, others report that severe covid causes 20 years of aging of the mmune system and others that covid makes one look visibly older. If the virus causes such dramatic aging, which supposedly is worsened with each reinfection, what do jabs and repeat jabs do?
I purchased 2 bottles 3 months ago. A bit spendy but it's an all in one for ease of adminstration. I have also followed flccc.net protocol and also Dr Zelenko, rip, for almost 2 years. Made little labeled pill pouches with everything in them for a my family members. Also spendy but they all had it on hand when they got covid. So for no one was hospitalized.